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논문 기본 정보

자료유형
학술저널
저자정보
임승준 (경남대학교) 신재숙 (경남대학교) 허만동 (경남대학교)
저널정보
한국체육과학회 한국체육과학회지 한국체육과학회지 제20권 제5호 (자연과학편)
발행연도
2011.10
수록면
1,139 - 1,150 (12page)

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초록· 키워드

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This study aims at understanding the effect of 8 weeks combined exercise training of body composition, metabolic syndrome risk factors, leptin, adiponetin and C-reactive Protein(CRP). 20 women were included in our study and 10 of them were randomly selected as a target group and the rest of them were selected as a control group. Combined exercise training had been carried out 8 weeks, and five days per week and once per day. The training is consist of 10 min. of warming up, 40~50 min of main training (treadmill: 20 min, dumbell and gymball: 20~30 min), and 10 min of cooling down, and the intensity of exercise is in 50~60% 1-RM and 50~60% HRmax. In order to analyze the differences by body composition, metabolic syndrome risk factors, hormone related to obesity and CRP, two way ANOVA was used. Conclusions of this study are as follows; 1. Changes of Body Composition: After 8 weeks combined exercise training weight, fat mass, BMI, and VFA decreased significantly and weight, % fat, fat mass, BMI, and VFA were affected interactively. 2. Changes in Risk Factors of Metabolic Syndrome: WHR, TG, SBP and DBP decreased significantly and SBP and DBP decreased significantly and interactive effect was shown. 3. Changes in Hormone Related to Obesity: After 8 weeks, Adiponectin increased significantly, and interactive effect was shown. 4. Changes in CRP: After 8 weeks, there was no significant change in CRP and no interactive effect. The study showed that 8 weeks combined exercise training not only deσeased the weight, fat percent, fat mass, BMI and VFA, but also reduced the risk factor of metabolic syndrome with having positive influences on obesity-related hormone and CRP which result would be expected to promote the health management for middle-aged women who are prone to be in failing health due to obesity that becomes an object of public concern along with other various causes.

목차

Abstract
Ⅰ. 서론
Ⅱ. 연구방법
Ⅲ. 결과
Ⅳ. 논의
Ⅴ. 결론
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UCI(KEPA) : I410-ECN-0101-2013-692-001072474