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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2004.1
- 수록면
- 193 - 203 (11page)
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A growing body of evidence, including studies using genetically engineered mouse models, has shown that Ca2+ cycling and Ca2+-dependent sig-naling pathways play a pivotal role in cardiac hypertrophy and heart failure. In addition, recent studies identified that mutations of the genes encoding sarcoplasmic reticulum (SR) proteins cause human cardiomyopathies and lethal ven-tricular arhythmias. The regulation of Ca2+ ho-via the SR proteins may have potential therapeutic value for heart diseases such as cardiomyopathy, heart failure and arrhythmias.
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등록된 정보가 없습니다.
참고문헌 (27)
참고문헌 신청
/ / Calcium cycling proteins in heart diseases 201
/ / Chronic phospholamban inhibition prevents progressive cardiac dysfunction and pathological remodeling after infarction in rats : 727 ~ 36
/ / Localization and partial characterization of the oligomeric disulfide-linked molecular weight 95 which binds the ryanodine and dihydropyridine receptors in skeletal muscl
/ 1997 / Overexpresion of the rat sarcoplasmic reticulum Ca2+ ATPase gene in the heart of transgenic mice accelerates calcium transients and cardiac relaxation : 380 ~ 9
/ 1998 / Pentameric assembly of phospholam-ban facilitates inhibition of cardiac function in vivo : 33674 ~ 80
/ / Chronic phospholamban inhibition prevents progressive cardiac dysfunction and pathological remodeling after infarction in rats : 727 ~ 36
/ / Localization and partial characterization of the oligomeric disulfide-linked molecular weight 95 which binds the ryanodine and dihydropyridine receptors in skeletal muscl
/ 1997 / Overexpresion of the rat sarcoplasmic reticulum Ca2+ ATPase gene in the heart of transgenic mice accelerates calcium transients and cardiac relaxation : 380 ~ 9
/ 1998 / Pentameric assembly of phospholam-ban facilitates inhibition of cardiac function in vivo : 33674 ~ 80
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