지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2015.1
- 수록면
- 41 - 45 (5page)
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초록· 키워드
오류제보하기
Chronic glomerulonephritis (GN), which includes focal segmental glomerulosclerosis and proliferative forms of GN such as IgA nephropathy, increases the risk of hypertension. Hypertension in chronic GN is primarily volume dependent,and this increase in blood volume is not related to the deterioration of renal function. Patients with chronic GN become salt sensitive as renal damage including arteriolosclerosis progresses and the consequent renal ischemia causes the stimulation of the intrarenal renin-angiotensin-aldosterone system(RAAS). Overactivity of the sympathetic nervous system also contributes to hypertension in chronic GN. According to the KDIGO guideline, the available evidence indicates that the target BP should be ≤140mmHg systolic and ≤90mmHg diastolic in chronic kidney disease patients without albuminuria. In most patients with an albumin excretion rate of ≥30mg/24 h (i.e., those with both micro- and macroalbuminuria), a lower target of ≤130mmHg systolic and ≤80mmHg diastolic is suggested. The use of agents that block the RAAS system is recommended or suggested in all patients with an albumin excretion rate of ≥30mg/24 h. The combination of a RAAS blockade with a calcium channel blocker and a diuretic may be effective in attaining the target BP, and in reducing the amount of urinary protein excretion in patients with chronic GN.
목차
등록된 정보가 없습니다.
참고문헌 (27)
참고문헌 신청
Nachman PH / 2011 / The kidney / Saunders :1101~1191
Orofino L / 1987 / Hypertension in primary chronic glomerulonephritis:analysis of 288 biopsied patients / Nephron 45:22~26
Csiky B / 1999 / Ambulatory blood pressure monitoring and progression in patients with IgA nephropathy / Nephrol Dial Transplant 14:86~90
Kaplan NM / 1998 / Clinical Hypertension / Williams & Wilkins :281~299
Joles JA / 2004 / Causes and consequences of increased sympathetic activity in renal disease / Hypertension 43:699~706
Orofino L / 1987 / Hypertension in primary chronic glomerulonephritis:analysis of 288 biopsied patients / Nephron 45:22~26
Csiky B / 1999 / Ambulatory blood pressure monitoring and progression in patients with IgA nephropathy / Nephrol Dial Transplant 14:86~90
Kaplan NM / 1998 / Clinical Hypertension / Williams & Wilkins :281~299
Joles JA / 2004 / Causes and consequences of increased sympathetic activity in renal disease / Hypertension 43:699~706
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