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학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
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논문 기본 정보
- 자료유형
- 학위논문
- 저자정보
- 지도교수
- 김민기
- 발행연도
- 2019
- 저작권
- 고려대학교 논문은 저작권에 의해 보호받습니다.
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초록· 키워드
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Normal body tissue or lesion characteristics in T1 images have been evaluated; however, how external parameters effect the change in signal intensity by gadolinium-based contrast agent remains unknown. We investigated how contrast enhancement changed according to echo time(TE) in 3.0T magnetic resonance (MR) T1 imaging and determined the optimal settings for TE in contrast-enhanced T1 imaging. Since there are no guidelines regarding parameters for T1 enhancement when using MR-contrast agents, we analyzed results from varying TEs (between 25 and 7 msec) in both a phantom and clinical study. We obtained the following results: contrast percentage of fat to saline increased from 740.0?1003.6%, response start point increased from 30?90 mmol, max peak signal intensity increased from 1771?2425 a.u., max peak point increased from 2?4 mmol, enhancement percentage of the max peak signal intensity (MPSI) to saline increased from 1671.0?2065.2%, the average of SI on each mol as TE increased from 600.8?996.6 a.u., the average of SI as TE on each molar concentration increased from 378?845 a.u., the AEPSS increased from 44.3?140.3%, and the AEPSC increased from 224.3?647.8%. We confirmed that TE can affect contrast enhancement, and the lowest TE has faster and higher effects on contrast enhancement.
목차
* Contents* Abstract1. Introduction ........................................................................................................... 12. Materials and Methods ........................................................................................ 62.1 Phantom study ................................................................................................ 62.1.1 Preparation of GBCA and the MR phantom ................................................... 62.1.2 MRI specification and protocol ...................................................................... 91) MRI specification ............................................................................................... 92) Sequence and parameters ................................................................................. 92.1.3 Experimental procedure ................................................................................ 102.2 Clinical study ................................................................................................... 122.3 Method of analysis .......................................................................................... 142.4 Statistical analysis ............................................................................................ 173. Results ................................................................................................................ 183.1 Phantom study ................................................................................................. 183.2 Change in GBCA SI ........................................................................................... 193.3 Clinical study ................................................................................................... 254. Discussion ........................................................................................................... 295. Conclusions .......................................................................................................... 366. References ........................................................................................................... 37
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