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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2015.1
- 수록면
- 231 - 235 (5page)
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초록· 키워드
오류제보하기
Damage to peripheral nerves or the spinal cord is often accompanied by neuropathic pain, which is a complex, chronic pain state. Increasing evidence indicates that alterations in the expression and activity of gap junction channels in the spinal cord are involved in the development of neuropathic pain. Thus, this review briefly summarizes evidence that regulation of the expression, coupling, and activity of spinal gap junction channels modulates pain signals in neuropathic pain states induced by peripheral nerve or spinal cord injury. We particularly focus on connexin 43 and pannexin 1 because their regulation vastly attenuates symptoms of neuropathic pain. We hope that the study of gap junction channels eventually leads to the development of a suitable treatment tool for patients with neuropathic pain. (Korean J Pain 2015; 28: 231-235)
목차
등록된 정보가 없습니다.
참고문헌 (25)
참고문헌 신청
Bethea JR / 2000 / Spinal cord injury-induced inflammation : a dual-edged sword / Prog Brain Res 128 : 33 ~ 42
Chen G / 2014 / Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice / Brain 137 : 2193 ~ 2209
Collo G / 1997 / Tissue distribution of the P2X7 receptor / Neuropharmacology 36 : 1277 ~ 1283
Cotrina ML / 2009 / Physiological and pathological functions of P2X7 receptor in the spinal cord / Purinergic Signal 5 : 223 ~ 232
De Vuyst E / 2007 / Connexin hemichannels and gap junction channels are differentially influenced by lipopolysaccharide and basic fibroblast growth factor / Mol Biol Cell 18
Chen G / 2014 / Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice / Brain 137 : 2193 ~ 2209
Collo G / 1997 / Tissue distribution of the P2X7 receptor / Neuropharmacology 36 : 1277 ~ 1283
Cotrina ML / 2009 / Physiological and pathological functions of P2X7 receptor in the spinal cord / Purinergic Signal 5 : 223 ~ 232
De Vuyst E / 2007 / Connexin hemichannels and gap junction channels are differentially influenced by lipopolysaccharide and basic fibroblast growth factor / Mol Biol Cell 18
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