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학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
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연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술대회자료
- 저자정보
- 발행연도
- 2021.7
- 수록면
- 89 - 89 (1page)
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초록· 키워드
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Objective: Brain arteriovenous malformations (BAVMs) occur in about 10% of hereditary hemorrhagic telangiectasia (HHT) patients and can cause life-threating intracranial hemorrhage. Longitudinal studies that allow monitoring the birth and progression of BAVMs would be tremendous not only for understanding the natural history of BAVMs but also for developing preclinical models.
Method: We used Tagln (SM22)-Cre:Alk1 <SUP>2f/2f</SUP> mice that were previously shown to develop BAVMs. GEFC (gradient echo with flow compensation) images were periodically acquired from the mutant and control mice starting at 1-2 months of age up to 14 months using Bruker 7T MR spectrometer. Tamoxifen inducible endothelial cell specific CreER (Scl -CreER) was used to determine critical stage of HHT gene deletion to cause BAVMs.
Result: Brains of 55 Tagln -Cre (+);Alk1 <SUP>2f/2f</SUP> mutant mice studied with magnetic resonance angiography were categorized into three groups: no de ... 전체 초록 보기
Method: We used Tagln (SM22)-Cre:Alk1 <SUP>2f/2f</SUP> mice that were previously shown to develop BAVMs. GEFC (gradient echo with flow compensation) images were periodically acquired from the mutant and control mice starting at 1-2 months of age up to 14 months using Bruker 7T MR spectrometer. Tamoxifen inducible endothelial cell specific CreER (Scl -CreER) was used to determine critical stage of HHT gene deletion to cause BAVMs.
Result: Brains of 55 Tagln -Cre (+);Alk1 <SUP>2f/2f</SUP> mutant mice studied with magnetic resonance angiography were categorized into three groups: no de ... 전체 초록 보기
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