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논문 기본 정보

자료유형
학술저널
저자정보
Xingyu Jia (epartment of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Songfeng Chen (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Qianjun Zhuang (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Niandi Tan (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Mengyu Zhang (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Yi Cui (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Jinhui Wang (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Xiangbin Xing (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China) Yinglian Xiao (Department of Gastroenterology The First Affiliated Hospital Sun Yat-sen University Guangzhou China)
저널정보
대한소화관운동학회(현 대한소화기능성질환.운동학회) Journal of Neurogastroenterology and Motility (JNM) Journal of Neurogastroenterology and Motility (JNM) Vol.29 No.2
발행연도
2023.4
수록면
145 - 155 (11page)
DOI
10.5056/jnm22176

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Achalasia is a primary esophageal motility disorder manifested by dysphagia and chest pain that impair patients’ quality of life, and it also leads to chronic esophageal inflammation by food retention and increases the risk of esophageal cancer. Although achalasia has long been reported, the epidemiology, diagnosis and treatment of achalasia are not fully understood. The current clinical dilemma of achalasia is mainly due to its unclear pathogenesis. In this paper, epidemiology, diagnosis treatment, as well as possible pathogenesis of achalasia will be reviewed and summarized. The proposed hypothesis on the pathogenesis of achalasia is that genetically susceptible populations potentially have a higher risk of infection with viruses, triggering autoimmune and inflammation responses to inhibitory neurons in lower esophageal sphincter.

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