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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2004.9
- 수록면
- 269 - 273 (5page)
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This study was designed to investigate the effect of ferulic acid on cell viability and cell adhesion activity in normal human gingival fibroblasts. The cell viability and cell adhesion activity of ferulic acid was measured by MTT assay or XTT assay, respectively, after normal human gingival fibroblasts were treated with or without ferulic acid for 48 hours. The cell viability of ferulic acid on normal human gingival fibroblasts did not show any decreasement by MTT assay and also, cell adhesion activity did not decreased by XTT assay, respectively, compared with control after cells were treated with various concentrations of ferulic acid for 48 hours. MTT?? and XTT?? were 2,130.0 μM and 1,773.7 μM ferulic acid, respectively. These results suggest that ferulic acid is non-toxic to normal human gingival fibroblasts by showing no significant differences in the cell viability and the adhesion activity compared with control by colorimetric assay.
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Ferulic acid prevents the injury-induced decrease of γ-enolase expression in brain tissue and HT22 cells
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Ferulic acid prevents the down-regulation of MEK/ERK/p90RSK signaling pathway in focal cerebral ischemic injury
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Ferulic Acid Prevents Middle Cerebral Artery Occlusion-induced Reduction in Thioredoxin Protein
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Cell viability analysis
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Toxic Effect on Phenolic Compound by Colorimeteric Assay in Normal NIH 3T3 Fibroblasts
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Ferulic acid ameliorates cerebral ischemic injury-induced reduction of hippocalcin expression
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수종의 저장용액에서의 치은섬유모세포 생존율의 비교연구
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2012 .01
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치은 섬유아세포 ( Gingival fibroblast )에 대한 저출력 레이저광의 효과에 관한 실험적 연구 ( AN EXPERIMENTAL STUDY ON THE EFFEITS OF LOW POWER DENSITY LASER ON THE HUMAN GINGIVAL FIBROBLAST )
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Cytotoxicity Evaluation of Elastomeric Impression Materials Using Different Fibroblasts Cell Lines
Journal of Korean Dental Science
2014 .12
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Ferulic acid regulates the AKT/GSK-3β/CRMP-2 signaling pathway in a middle cerebral artery occlusion animal model
Laboratory Animal Research
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UCI(KEPA) : I410-ECN-0101-2009-510-017341808