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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2017.1
- 수록면
- 506 - 519 (14page)
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초록· 키워드
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The “Barker hypothesis” postulates that a number of organ structures and associated functions undergo programming during embryonic and fetal life, which determines the set point of physiological and metabolic responses that carry into adulthood. Hence, any stimulus or insult at a critical period of embryonic and fetal development can result in developmental adaptations that produce permanent structural, physiological and metabolic changes, thereby predisposing an individual to cardiovascular, metabolic and endocrine disease in adult life. This article will provide evidence linking these diseases to fetal undernutrition and an overview of previous studies in this area as well as current advances in understanding the mechanism and the role of the placenta in fetal programming.
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참고문헌 (30)
참고문헌 신청
Barker DJ / 1993 / Type 2 (non-insulin-dependent) diabetes mellitus, hypertension and hyperlipidaemia (syndrome X): relation to reduced fetal growth / Diabetologia 36 : 62 ~
Barker DJ / 1995 / Fetal origins of coronary heart disease / BMJ 311 : 171 ~ 174
Barker DJ / 1997 / Maternal nutrition, fetal nutrition, and disease in later life / Nutrition 13 : 807 ~ 813
Barker DJ / 1998 / In utero programming of chronic disease / Clin Sci (Lond) 95 : 115 ~ 128
Barker DJ / 1998 / Mothers, babies, and health in later life / Churchill Livingstone
Barker DJ / 1995 / Fetal origins of coronary heart disease / BMJ 311 : 171 ~ 174
Barker DJ / 1997 / Maternal nutrition, fetal nutrition, and disease in later life / Nutrition 13 : 807 ~ 813
Barker DJ / 1998 / In utero programming of chronic disease / Clin Sci (Lond) 95 : 115 ~ 128
Barker DJ / 1998 / Mothers, babies, and health in later life / Churchill Livingstone
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