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학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
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연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
초록· 키워드
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Atopic dermatitis (AD) and psoriasis are inflammatory skin disorders that impose considerable burdens upon patient’s quality of life, health outcome, and work productivity. Atopic dermatitis involves erythematous patches that are not well demarcated and are often excoriated and associated with severe pruritus. Psoriasis presents with well-demarcated plaques covered with silvery white scales with variable levels of pruritus. Despite important clinical distinctions, the two disorders are similar in that epidermal keratinocytes respond to T-cell derived cytokines by altering growth and differentia tion responses, accounting for major parts of the overall dis ease phenotype. Psoriasis is a disease largely driven by Th17 T-cells and associated IL-17 activation, while AD has a strong Th2 component associated with IL-4, IL-13 over-pro duction, and both disorders have activation of Th22 T-cells and Th1 pathways with increased IL-22 and IFN-γ produc tion. AD is a disease frequently associated with increased IgE production and overt allergies or asthma, most likely due to increased Th2 activation, which is largely lacking in psoria sis. In, this article, I will review and compare the immuno pathogenesis of AD and psoriasis.
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