지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2024.9
- 수록면
- 109 - 118 (10page)
- DOI
- 10.25289/ML.24.023
이용수
초록· 키워드
오류제보하기
Mitochondrial dysfunction is a critical factor in the pathogenesis of neurodegenerative diseases such as Alzheimer’s, Parkinson’s, and amyotrophic lateral sclerosis. Disruptions in mitochondrial fusion and fission lead to the accumulation of damaged mitochondria and elevated oxidative stress, contributing to neuronal cell death. Photobiomodulation (PBM) therapy, utilizing low-level laser light or light-emitting diodes, has shown promise in restoring mitochondrial dynamics by stimulating cytochrome c oxidase in the mitochondrial respiratory chain. This enhances adenosine triphosphate production, promotes mitochondrial fusion, and reduces fission. Additionally, PBM attenuates neuroinflammation by reducing pro-inflammatory cytokine and reactive oxygen species production, creating a favorable environment for neuronal survival. This review explores the mechanisms through which PBM influences mitochondrial dynamics and its therapeutic potential in neurodegenerative diseases. By restoring mitochondrial balance and reducing neuroinflammation, PBM offers a unique approach to mitigating mitochondrial dysfunction and enhancing neuronal function.
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