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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2017.1
- 수록면
- 74 - 76 (3page)
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Dear Editor, There are three types of BCR-ABL1 fusion transcripts that differ on the basis of the breakpoint in the BCR gene [1]. The major type, encoding p210, has two isoforms, b2a2 (e13a2) and b3a2 (e14a2), and is associated with more than 95% of Ph-positive CML cases. The minor type, encoding p190, is e1a2. The micro type, encoding p230, is e19a2. This micro type is rare, and has been associated with a variety of hematologic malignancies including neutrophilic CML (CML-N), all phases of classical CML, essential thrombocythemia (ET), and acute myeloid or lymphoblastic leukemia [2, 3]. Here, we describe the first case of chronic phase CML concurrently expressing e1a2 and e19a2 BCR-ABL1.
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참고문헌 (9)
참고문헌 신청
Bertorelle R / 2001 / The e19a2 BCR/ABL fusion transcript with additional chromosomal aberrations on a new case of chronic myeloid leukemia (CML) of mild type / Leukemia 15 :
Briz M / 1997 / Typical chronic myelogenous leukemia with e19a2 junction BCR/ABL transcript / Blood 90 : 5024 ~ 5025
How GF / 1998 / Chronic myeloid leukemia with e19a2 (c3a2) BCR/ABL fusion junction--is it truly a benign disease? / Leukemia 12 : 1166 ~ 1167
Jeon EK / 2011 / The first case of acute lymphoblastic leukemia with the e19a2 BCR-ABL1 transcript: imatinib therapy followed by unrelated donor transplantation induces a dur
Melo JV / 1996 / The diversity of BCR-ABL fusion proteins and their relationship to leukemia phenotype / Blood 88 : 2375 ~ 2384
Briz M / 1997 / Typical chronic myelogenous leukemia with e19a2 junction BCR/ABL transcript / Blood 90 : 5024 ~ 5025
How GF / 1998 / Chronic myeloid leukemia with e19a2 (c3a2) BCR/ABL fusion junction--is it truly a benign disease? / Leukemia 12 : 1166 ~ 1167
Jeon EK / 2011 / The first case of acute lymphoblastic leukemia with the e19a2 BCR-ABL1 transcript: imatinib therapy followed by unrelated donor transplantation induces a dur
Melo JV / 1996 / The diversity of BCR-ABL fusion proteins and their relationship to leukemia phenotype / Blood 88 : 2375 ~ 2384
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